Posts from March 2015 (48)

Team Ratings for Round 4

The basic method is described on my Department home page.

Here are the team ratings prior to this week’s games, along with the ratings at the start of the season.

Performance So Far

So far there have been 24 matches played, 16 of which were correctly predicted, a success rate of 66.7%.

Here are the predictions for last week’s games

Predictions for Round 4

Here are the predictions for Round 4. The prediction is my estimated expected points difference with a positive margin being a win to the home team, and a negative margin a win to the away team.

Team Ratings for Round 7

The basic method is described on my Department home page.

Here are the team ratings prior to this week’s games, along with the ratings at the start of the season.

Performance So Far

So far there have been 40 matches played, 26 of which were correctly predicted, a success rate of 65%.

Here are the predictions for last week’s games.

Predictions for Round 7

Here are the predictions for Round 7. The prediction is my estimated expected points difference with a positive margin being a win to the home team, and a negative margin a win to the away team.

• From Dylan Cleaver and Harkanwal Singh at the Herald, an interactive graphic for exploring comparisons between players over the history of one-day internationals.
• From the Herald Data blog, or with interactive versions at his website, a visualisation by Michael Lascarides of how each innings progressed. Mostly this is for whole teams,but here’s Martin Guptill’s double century against the West Indies

Most stories about population genetic ancestry tend to be based on pure male-line or pure female-line ancestry, which can be unrepresentative.  That’s especially true when you’re looking at invasions — invaders probably leave more Y-chromosomes behind than the rest of the genome.  There’s a new UK study that used data on the whole genome from a few thousand British people, chosen because all four of their grandparents lived close together.  The idea is that this will measure population structure at the start of the twentieth century, before people started moving around so much.

Here’s the map of ancestry clusters. As the story in the Guardian explains, one thing it shows that the Romans and Normans weren’t big contributors to population ancestry, despite their impact on culture.

The “It’s not paranoia if..” issue

• A new initiative, Data Justice, concerned with widespread commercial data collection and analysis as a threat to privacy and equality.

• Some of the reasons that data-based decision making goes bad

• Trying to get “open” data in New Jersey: “initially refused to answer The Jersey Journal’s OPRA request because it didn’t make it on the agency’s standardized OPRA form, which wasn’t available on the NBHA website. Even after a reporter noted that in 2009 the state Supreme Court ruled standardized forms aren’t necessary, Earl wouldn’t accept a request on anything but the agency’s form.”

• Reidentification of ‘anonymised’ data: ‘When data is released after applying current ad-hoc de-identification methods, the privacy risks of re-identification are not just unknown but unknowable.’

Each week, we would like to invite readers of Stats Chat to submit nominations for our Stat of the Week competition and be in with the chance to win an iTunes voucher.

Here’s how it works:

• Anyone may add a comment on this post to nominate their Stat of the Week candidate before midday Friday March 27 2015.
• Statistics can be bad, exemplary or fascinating.
• The statistic must be in the NZ media during the period of March 21 – 27 2015 inclusive.
• Quote the statistic, when and where it was published and tell us why it should be our Stat of the Week.

Next Monday at midday we’ll announce the winner of this week’s Stat of the Week competition, and start a new one.

(more…)

If you’d like to comment on or debate any of this week’s Stat of the Week nominations, please do so below!

One way medical statisticians are trained into skepticism over their careers is seeing all the exciting ideas from excited scientists and clinicians that don’t turn out to work. Looking at old hypotheses is a good way to start. This graph is from a 1986 paper in the journal Medical Hypotheses, and the authors are suggesting pork consumption is important in multiple sclerosis, because there’s a strong correlation between rates of multiple sclerosis and pork consumption across countries:

This wasn’t a completely silly idea, but it was never anything but suggestive, for two main reasons. First, it’s just a correlation. Second, it’s not even a correlation at the level of individual people — the graph is just as strong support for the idea that having neighbours who eat pork causes multiple sclerosis. Still, dietary correlations across countries have been useful in research.

If you wanted to push this idea today, as a Twitter account claiming to be from a US medical practice did, you’d want to look carefully at the graph rather than just repeating the correlation. There are some countries missing, and other countries that might have changed over the past three decades.

In particular, the graph does not have data for Korea, Taiwan, or China. These have high per-capita pork consumption, and very low rates of multiple sclerosis — and that’s even more true of Hong Kong, and specifically of Chinese people in Hong Kong.  In the other direction, the hypothesis would imply very low levels of multiple sclerosis among US and European Jews. I don’t have data there, but in people born in Israel the rate of multiple sclerosis is moderate among those of Ashkenazi heritage and low in others, which would also mess up the correlations.

You might also notice that the journal is (or was) a little non-standard, or as it said  “intended as a forum for unconventional ideas without the traditional filter of scientific peer review”.

Most of this information doesn’t even need a university’s access to scientific journals — it’s just out on the web.  It’s a nice example of how an interesting and apparently strong correlation can break down completely with a bit more data.

I posted a version of this graph with ten years of weekly data, and Mark Stockdale pointed out there are quarterly data back to 1983 (isn’t official data wonderful?). You’ll need to click the graph to embiggen for easy viewing.

The horizontal axis is the import cost plus freight and insurance (with CPI adjustments to 2013 NZ dollars), and the vertical axis is the importer margin, which covers transport and sale costs within New Zealand, and profit. The idea is that local costs are typically slowly varying, so that short-term variation in margin tracks short-term variation in profit. The label for each year is on the data point for June.

The import cost plummeted in the early 1980s, soon followed by a drop in the importer margin. That’s presumably Rogernomics and its consequences. The cost stayed fairly stable and low in the 1990s and the margin drifted down.  Then the cost increased after 1999, with the margin staying stable. We’ve recently entered a new pattern, with margin drifting upwards.

A final note: the import cost is about the same as in 1983, and so is the retail price (in real terms). The reduction in importer margin since 1983 has been almost exactly matched by an increase in taxes, though the taxes would probably be higher under a realistic world carbon price.

The flame retardant chemicals in your phone made zebra fish “chubby”, says the caption on this photo at news.com.au. Zebra fish, as it explains, are a common model organism for medical research, so this could be relevant to people

On the other hand, as @LewSOS points out on Twitter, it doesn’t seem to be having the same effect on the model organisms in the photo.

What’s notable about the story is how much better it is than the press release, which starts out

Could your electronics be making you fat? According to University of Houston researchers, a common flame retardant used to keep electronics from overheating may be to blame.

The news.com.au story carefully avoids repeating this unsupported claim.  Also, the press release doesn’t link to the research paper, or even say where it was published (or even that it was published). That’s irritating in the media but unforgivable in a university press release.   When you read the paper it turns out the main research finding was that looking at fat accumulation in embryonic zebrafish (which is easy because they are transparent, one of their other advantages over mice) was a good indication of weight gain later in life, and might be a useful first step in deciding which chemicals were worth testing in mice.

So, given all that, does your phone or computer actually expose you to any meaningful amount of this stuff?

The compounds in question, Tetrabromobisphoneol A (TBBPA) and tetrachlorobisphenol A (TCBPA) can leach out of the devices and often end up settling on dust particles in the air we breathe, the study found.

That’s one of the few mistakes in the story: this isn’t what the study found, it’s part of the background information. In any case, the question is how much leaches out. Is it enough to matter?

The European Union doesn’t think so

The highest inhalation exposures to TBBP-A were found in the production (loading and mixing) of plastics, with 8-hour time-weighted-averages (TWAs) up to 12,216 μg/m³ . At the other end of the range, offices containing computers showed TBBP-A air concentrations of less than 0.001 μg/m³ . TBBP-A exposures at sites where computers were shredded, or where laminates were manufactured ranged from 0.1 to 75 μg/m³ .

You might worry about the exposures from plastics production, and about long-term environmental accumulations, but it looks like TBBP-A from being around a phone isn’t going to be a big contributor to obesity. That’s also what the international comparisons would suggest — South Korea and Singapore have quite a lot more smartphone ownership than Australia, and Norway and Sweden are comparable, all with much less obesity.